Omurilik Acillerinde Anestezi Yönetimi

Özet

Acil omurilik yaralanmaları, yüksek mortalite ve morbidite oranları nedeniyle dünya genelinde kritik bir sağlık sorunudur. Bu vakaların anestezi yönetimi; havayolu stabilizasyonu, hemodinamik dengenin korunması ve nöromonitorizasyon sinyallerinin doğru yorumlanması gibi zorlukları beraberinde getirir. C3-C5 seviyesindeki hasarlar diyafram fonksiyonunu bozarak solunum yetmezliğine yol açarken, T1-T4 seviyesindeki yaralanmalar vagal bradikardi ve nörojenik şoka neden olabilir. Preoperatif dönemde radyolojik inceleme ve nörolojik değerlendirme hayati önem taşır. Entübasyon sırasında servikal omurganın korunması için aşırı hareketten kaçınılmalı, ciddi yaralanmalarda uyanık fiberoptik entübasyon tercih edilmelidir. İntraoperatif süreçte, nöromonitorizasyon kullanılacaksa sinyalleri etkilemeyen total intravenöz anestezi (TIVA) önerilir. Hemodinamik yönetimde ortalama arter basıncının 50 mmHg üzerinde tutulması ve uygun vazopresör kullanımı sekonder hasarları önlemek için kritiktir. Ayrıca, kronik dönemdeki T7 üzeri yaralanmalarda otonom hiperrefleksi riskine karşı derin anestezi uygulanmalıdır. Postoperatif süreçte ise laringofaringeal ödem gibi havayolu komplikasyonlarına hazırlıklı olunmalı ve multimodal ağrı tedavisi planlanmalıdır.

Emergency spinal cord injuries are a critical global health issue due to high mortality and morbidity rates. Anesthesia management of these cases involves challenges such as airway stabilization, maintaining hemodynamic balance, and correct interpretation of neuromonitoring signals. Injuries at the C3-C5 level cause respiratory failure by impairing diaphragm function, while injuries at the T1-T4 level can lead to vagal bradycardia and neurogenic shock. Radiological examination and neurological assessment are vital in the preoperative period. During intubation, excessive movement must be avoided to protect the cervical spine, and awake fiberoptic intubation is preferred in severe injuries. During the intraoperative process, if neuromonitoring is to be used, total intravenous anesthesia (TIVA), which does not affect signals, is recommended. In hemodynamic management, maintaining mean arterial pressure above 50 mmHg and using appropriate vasopressors are critical to prevent secondary damage. Additionally, deep anesthesia should be applied against the risk of autonomic hyperreflexia in injuries above T7 in the chronic period. In the postoperative period, one should be prepared for airway complications such as laryngopharyngeal edema, and multimodal pain treatment should be planned.

Referanslar

Davis JW, Phreaner DL, Hoyt DB, Mackersie RC. The etiology of missed cervical spine injuries. J Trauma. 1993;34(3):342-6.

Stauffer ES, Bell GD. Traumatic respiratory quadriplegia and pentaplegia. Orthop Clin North Am. 1978;9(4):1081-9.

Theodore J, Robin ED. Speculations on neurogenic pulmonary edema (NPE). Am Rev Respir Dis. 1976;113(4):405-11.

Hawryluk G, Whetstone W, Saigal R, Ferguson A, Talbott J, Bresnahan J, et al. Mean Arterial Blood Pressure Correlates with Neurological Recovery after Human Spinal Cord Injury: Analysis of High Frequency Physiologic Data. J Neurotrauma. 2015;32(24):1958-67.

Bao FP, Zhang HG, Zhu SM. Anesthetic considerations for patients with acute cervical spinal cord injury. Neural Regen Res. 2017;12(3):499-504.

Martini RP, Larson DM. Clinical evaluation and airway management for adults with cervical spine instability. Anesthesiol Clin. 2015;33(2):315-27.

Como JJ, Diaz JJ, Dunham CM, Chiu WC, Duane TM, Capella JM, et al. Practice management guidelines for identification of cervical spine injuries following trauma: update from the eastern association for the surgery of trauma practice management guidelines committee. J Trauma. 2009;67(3):651-9.

Robitaille A. Airway management in the patient with potential cervical spine instability: continuing professional development. Can J Anaesth. 2011;58(12):1125-39.

Abd-Elsayed AA, Farag E. Anesthesia for cervical spine surgery. In: Farag E, editor. Anesthesia for Spine Surgery. Cambridge: Cambridge University Press; 2012. p. 178-87.

Turkstra TP, Craen RA, Pelz DM, Gelb AW. Cervical spine motion: a fluoroscopic comparison during intubation with lighted stylet, GlideScope, and Macintosh laryngoscope. Anesth Analg. 2005;101(3):910-5.

Donaldson 3rd W, Towers JD, Doctor A, Brand A, Donaldson VP. A methodology to evaluate motion of the unstable spine during intubation techniques. Spine. 1993;18(14):2020-3.

Keller C, Brimacombe J, Keller K. Pressures exerted against the cervical vertebrae by the standard and intubating laryngeal mask airways: a randomized, controlled, cross-over study in fresh cadavers. Anesth Analg. 1999;89(5):1296-300.

Robitaille A, Williams SR, Tremblay MH, Guilbert F, Thériault M, Drolet P. Cervical spine motion during tracheal intubation with manual in-line stabilization: direct laryngoscopy versus GlideScope videolaryngoscopy. Anesth Analg. 2008;106(3):935-41, table of contents.

Hindman BJ, Santoni BG, Puttlitz CM, From RP, Todd MM. Intubation biomechanics: laryngoscope force and cervical spine motion during intubation with Macintosh and Airtraq laryngoscopes. Anesthesiology. 2014;121(2):260-71.

Brimacombe J, Keller C, Künzel KH, Gaber O, Boehler M, Pühringer F. Cervical spine motion during airway management: a cinefluoroscopic study of the posteriorly destabilized third cervical vertebrae in human cadavers. Anesth Analg. 2000;91(5):1274-8.

Tamkus AA, Rice KS, Kim HL. Differential rates of false-positive findings in transcranial electric motor evoked potential monitoring when using inhalational anesthesia versus total intravenous anesthesia during spine surgeries. Spine J. 2014;14(8):1440-6.

Heath K, Erskine R. The anaesthetic management of spinal injuries and surgery to the cervical spine. Textbook of neuroanaesthesia and critical care Greenwich Medical Media, London. 2000:241-52.

Shaikh N, Rhaman MA, Raza A, Shabana A, Malstrom MF, Al-Sulaiti G. Prolonged bradycardia, asystole and outcome of high spinal cord injury patients: Risk factors and management. Asian J Neurosurg. 2016;11(4):427-32.

Sharma HS. Pathophysiology of blood-spinal cord barrier in traumatic injury and repair. Curr Pharm Des. 2005;11(11):1353-89.

Squair JW, Bélanger LM, Tsang A, Ritchie L, Mac-Thiong JM, Parent S, et al. Spinal cord perfusion pressure predicts neurologic recovery in acute spinal cord injury. Neurology. 2017;89(16):1660-7.

Saadeh YS, Smith BW, Joseph JR, Jaffer SY, Buckingham MJ, Oppenlander ME, et al. The impact of blood pressure management after spinal cord injury: a systematic review of the literature. Neurosurg Focus. 2017;43(5):E20.

Winter SF, Santaguida C, Wong J, Fehlings MG. Systemic and Topical Use of Tranexamic Acid in Spinal Surgery: A Systematic Review. Global Spine J. 2016;6(3):284-95.

Harrois A, Hamada SR, Duranteau J. Fluid resuscitation and vasopressors in severe trauma patients. Curr Opin Crit Care. 2014;20(6):632-7.

Fraser A, Edmonds-Seal J. Spinal cord injuries. A review of the problems facing the anaesthetist. Anaesthesia. 1982;37(11):1084-98.

Raeder JC, Gisvold SE. Perioperative autonomic hyperreflexia in high spinal cord lesions: a case report. Acta Anaesthesiol Scand. 1986;30(8):672-3.

Erickson RP. Autonomic hyperreflexia: pathophysiology and medical management. Arch Phys Med Rehabil. 1980;61(10):431-40.

Sagi HC, Beutler W, Carroll E, Connolly PJ. Airway complications associated with surgery on the anterior cervical spine. Spine (Phila Pa 1976). 2002;27(9):949-53.

Calder I. Anaesthesia for spinal surgery. In: Matta BF, Menon DK, Smith M, editors. Core Topics in Neuroanaesthesia and Neurointensive Care. Cambridge: Cambridge University Press; 2011. p. 222-36.

Sayfalar

297-304

Gelecek

11 Nisan 2022

Lisans

Lisans